Actinogen Medical Announces Landmark Research on Excess Cortisol and Alzheimer’s Disease
July 27 2016 - 1:30PM
Business Wire
- Australian CSIRO and university
funded AIBL study links excess cortisol and Alzheimer’s
disease.
- Anti-Alzheimer’s compound Xanamem™
in clinical testing blocks high cortisol (“stress” hormone)
associated with disease progression.
- Landmark research on disease
mechanism and Actinogen Medical’s Xanamem™ presented at Alzheimer’s
Association International Conference (AAIC) in Toronto 22nd-28th
July.
Actinogen Medical (ASX:ACW) announced today results of its lead
compound, Xanamem™, a novel, orally administered 11β-HSD1
inhibitor, which demonstrated in a Phase I study that it
significantly inhibited production of cortisol in healthy
volunteers and successfully crossed the blood-brain barrier. A
separate study, the Australian Imaging, Biomarker & Lifestyle
Flagship Study of Ageing (AIBL), sponsored by the CSIRO and a
number of Australian universities, showed a correlation between
elevated cortisol in the blood of a healthy aged population and the
subsequent development of Alzheimer’s disease in these individuals.
When individuals also evidenced a broad build-up of beta-amyloid
plaques in the brain, their chances of developing Alzheimer’s
disease increased even further. The AIBL study (n=416) concluded
that targeting ways to lower excess cortisol should be undertaken
in battling Alzheimer’s disease in the elderly. Both studies were
presented at the AAIC, the world’s largest Alzheimer’s Dementia
meeting, taking place this week in Toronto, Canada.
Professor Jeffrey Cummings, M.D., Director, Cleveland Clinic Lou
Ruvo Center for Brain Health, United States commented, “These study
results demonstrate both the importance of understanding the
pathological processes in Alzheimer’s and the compelling need for
new approaches to treatment. To my eyes, AIBL has provided the most
important validation to date for controlling excess cortisol
production in individuals at risk for developing dementia.
Development of new therapies to inhibit cortisol can show us the
impact of blocking this mechanism on disease progression.”
In 2016, Actinogen Medical initiated XanADu, the Company’s
pivotal global Phase II clinical trial investigating Xanamem™ as a
treatment for mild Alzheimer’s disease. Xanamem™ has been
specifically designed to inhibit the excess production of cortisol,
the “stress hormone,” in the brain. XanADu is being conducted at
trial sites in the US, Australia, and the UK, with the primary
efficacy endpoints of improvements in scores on the Alzheimer’s
Disease Composite (ADCOMs) and Alzheimer's Disease Assessment
Scale-Cognitive (ADAS-Cog) version 14 tests. The clinical trial is
expected to enroll patients in 2H16.
“The findings from the AIBL study, linking excess cortisol with
the development of Alzheimer’s disease, provides further strong
validation of our ongoing development of Xanamem™,” said Dr. Bill
Ketelbey, CEO of Actinogen Medical. “Independent validation is
clearly emerging that excess cortisol is a key target for treating
the disease and our XanADu trial aims to demonstrate that
inhibiting cortisol in the brain with Xanamem™ is an effective
treatment option for patients with mild Alzheimer’s disease. It’s
particularly exciting to receive this endorsement of Xanamem™’s
novel mechanism of action as Alzheimer’s is a disease where new
approaches to its management are desperately needed to help
millions of people worldwide.”
Data Presentations at the AAIC Conference
Xanamem™“Xanamem™ a novel 11β–HSD1 inhibitor with
potential to provide durable symptomatic and disease modifying
benefits in Alzheimer’s disease” (Abstract No. a12215)Timing:
1:00pm Eastern time, Wednesday, July 27, 2016Location: P4:388
Professor Craig Ritchie, MD, Ph.D., Chair of the Psychiatry of
Ageing and Director of the Centre for Dementia Prevention, The
University of Edinburgh, United Kingdom and author on the Xanamem™
study, commented, “In the study, we saw clear signals of an effect
through the substantial inhibition of extra-adrenal regeneration of
cortisol. We are also excited to see clear evidence of delivery
through the blood-brain barrier, which we believe has hampered
programs targeting this mechanism in the past. We believe this is a
promising compound, and we are excited to see the progress into
Phase II testing.”
Australian Imaging, Biomarker & Lifestyle Flagship Study
of Ageing (AIBL)“Plasma cortisol, amyloid-β and cognitive
decline in preclinical Alzheimer’s disease.” (Abstract No.
a10218)Timing: 1:00pm Eastern time, Tuesday, July 26, 2016Location:
P3:308
Professor Colin L. Masters, M.D., Co-Head, Neurodegeneration
Division of the Florey Institute of Neuroscience and Mental Health,
Melbourne, Australia, and co-author on the AIBL study, stated,
“While the presence of aberrant beta-amyloid and tau proteins in
the brain, combined with neural death and cognitive decline, are
recognized as the hallmarks of Alzheimer’s disease, there is still
substantial speculation as to the underlying triggers for the
disease. We have demonstrated that when levels of cortisol, the
‘stress hormone,’ become chronically elevated in the blood, there
is a strong correlation with the subsequent development of
Alzheimer’s disease. This finding, and the results showing synergy
with the build-up of beta-amyloid plaques in the brain, suggests a
compelling new area of research for the treatment of
Alzheimer’s.”
For information on both presentations you can visit the
following links: http://bit.ly/AIBLposter and
http://bit.ly/XanamemPoster.
About Xanamem™
Xanamem™ is being developed as a promising new therapy for
Alzheimer’s disease, a condition with a multi- billion dollar
market potential. The cost of Alzheimer’s treatment in the US alone
was estimated to be US$250bn in 2013, with this cost estimated to
increase to US$1 trillion by 2050, outstripping the cost of
treating all other diseases. Alzheimer’s disease is now the second
leading cause of death in Australia behind ischaemic heart disease.
Xanamem™’s novel mechanism of action sets it apart from existing
Alzheimer’s treatments. It works by blocking the production of
cortisol - the stress hormone - in the hippocampus and frontal
cortex, the areas of the brain most affected by Alzheimer’s
disease. There is growing evidence that chronic stress and elevated
cortisol levels lead to changes in the brain affecting memory and
to the development of amyloid plaques and neural death – the
hallmarks of Alzheimer’s disease.
About Actinogen Medical
Actinogen Medical is focused on an innovative approach, through
the inhibition of excess cortisol production, for treating
cognitive impairment in chronic neurodegenerative and metabolic
diseases. The lead candidate drug Xanamem™, blocks the development
of cortisol which appears to be associated with cognitive
impairment and the development of amyloid plaques and neural death
in the brain – the hallmarks of Alzheimer’s disease. In 2016 the
Company initiated XanADu, a Phase II efficacy and safety trial in
mild Alzheimer’s disease.
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version on businesswire.com: http://www.businesswire.com/news/home/20160727006263/en/
Company:Actinogen MedicalDr. Bill Ketelbey, +61 2
8964 7401CEO & Managing
Directorbill.ketelbey@actinogen.com.au@BillKetelbeyorMedia:Burns
McClellanJustin Jackson,
+1-212-213-0006jjackson@burnsmc.com@BurnsPR
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