Cardium Provides Update on Commercial Development Plans for Generx Angiogenic Therapy for Heart Disease at 2010 Cell & Gene Ther
January 26 2010 - 9:00AM
PR Newswire (US)
SAN DIEGO, Jan. 26 /PRNewswire-FirstCall/ -- Cardium Therapeutics
(NYSE Amex: CXM) reported today that its Cardium Biologics division
provided an update on plans for the continuing commercial
development of Generx(TM) (alferminogene tadenovec, Ad5FGF-4), a
DNA-based angiogenic therapy product candidate for patients with
coronary artery disease. The update was presented by Gabor M.
Rubanyi, M.D., Ph.D., Cardium's Chief Scientific Officer at the
annual 2010 Cell & Gene Therapy Forum in Washington, D.C. on
January 25, 2010. An investor presentation that includes material
from Dr. Rubanyi's presentation is now available on Cardium's
website at
http://phx.corporate-ir.net/phoenix.zhtml?c=77949&p=irol-presentations.
(Logo: http://www.newscom.com/cgi-bin/prnh/20051018/CARDIUMLOGO)
Cardium Biologics reported on the following findings and plans: (1)
As previously announced, based on an agreement with the FDA, Generx
would be re-formulated to increase its shelf life, and further
formulation enhancements are expected to allow for storage using a
standard freezer (rather than at -70 degrees C), and potentially a
lyophilized version for refrigerated storage. (2) Based on clinical
and pre-clinical findings, angiogenic therapy appears to lead to
long-term functional improvements in cardiac microvascular
circulation, and Cardium believes that cardiac perfusion (as
measured by SPECT) appears to be an important efficacy endpoint to
consider that is now supported by a 10-year study of the
cardio-protective nature of collateral circulation (Meier et al.
Circulation 2007; 116:975-83). (3) Recent preclinical data
developed by the Company under an SBIR grant indicate that
localized ischemia can significantly increase Ad5 transfection in
the heart in association with intracoronary infusion therapy,
suggesting that cardiac stimulation (using e.g. dobutamine which is
routinely administered to patients) in association with
administration of Generx has the potential to dramatically enhance
the therapeutic effect at a given dose. (4) In order to incorporate
these enhanced formulations and perspectives and also expand the
clinical database supporting the potential commercialization of
Generx, the Company is considering clinical and commercialization
pathways for Generx in developing nations that often have very
limited access to surgical approaches such as angioplasty, stenting
or coronary artery bypass (which are available to patients in
industrialized nations but at costs reaching $80,000 to $100,000
per procedure over a five year period). The U.S. and European
clinical studies of Generx have collectively established an
extensive database supporting the safety of this product candidate,
even in patients with severe forms of chronic coronary artery
disease. Successful application of this therapy as "front-line
care" would be expected to lead to commercialization pathways in
many developing markets for which surgical and other relatively
expensive approaches are either unavailable or limited by already
over-burdened health care systems. In addition, information gained
with these new applications would be expected to be useful to
support broadened commercialization pathways in the U.S. and other
developed markets. "There has been significant progress in the care
and treatment of patients with cardiovascular disease in the
industrialized world, although at considerable and increasing
expense. While heart disease remains a very serious problem in the
U.S. and Europe, incidence is rapidly increasing in large parts of
the newly-industrializing world such as China, India and Russia, as
well as in the Middle East and Latin America. In many countries,
healthcare systems are unable to provide wide access to relatively
expensive procedures such as coronary angioplasty, stenting, or
cardiac bypass surgery. Based on data indicating that the Generx
product candidate appears to safe and has the potential to
substantially increase coronary blood flow in the context of heart
disease - coupled with recent findings that improvements in
collateral circulation are associated with long-term benefits,
including reduced mortality as reported in a 10-year study
published in Circulation - we believe that this product candidate
could be developed as a front-line therapy for coronary artery
disease. Additional data gained in parallel studies, which would be
expected to be conducted in collaboration with specific regional
development partners, would also be expected to support an expanded
U.S. registration dossier by providing additional safety data and
potentially alternative efficacy measures," stated Christopher J.
Reinhard, Cardium Chairman and Chief Executive Officer. Mechanism
of Action Study: Generx Improves Blood Flow via Increased
Collateral Circulation in Ischemic Heart Tissue Reported in Journal
of American College of Cardiology Positive results supporting the
proposed mechanism of action of Generx in improving blood flow
within ischemic heart tissue were published in JACC, A Randomized,
Double-Blind, Placebo-Controlled Trial of Ad5FGF-4 Gene Therapy and
its Effect on Myocardial Perfusion in Patients with Stable Angina
(Grines et al., J Am Coll Cardiol 2003; 42:1339-47). The results of
the study showed improvements in myocardial perfusion (blood flow)
in the ischemic region of the hearts of both men and women
following a single intracoronary infusion of Generx. Increases in
blood flow within the ischemic regions of the heart under the
conditions studied (i.e., adenosine infusion) are believed to be
due to the formation of new collateral blood vessels capable of
providing additional blood flow capacity under stress. Generx was
well tolerated in the study of 52 patients (88% men and 22% women)
with reversible ischemia of >9%, with no adverse sequelae. As
noted in the publication, the mean change observed in
Generx-treated patients was a 4.2% absolute reduction (which
represents a 20% relative reduction) in the reversible perfusion
defect size from baseline at eight weeks (p